Diet plays an important role for kidney disease patients, it can help reduce kidney burden and slow down the disease. Have you got a good diet chart with your kidney disease? This blog can help you get further understanding.

Pathogenesis of Hypertensive Nephropathy


Hypertensive nephropathy is always the cause of concern. Mainly because of the fear of hypertensive nephropathy and the current treatment of hypertensive nephropathy, people who have not yet sick who want the disease do not happen to themselves, and have been sick people are thinking about how better For treatment. There are many types of kidney disease, hypertensive nephropathy is one of them.

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Experts pointed out: Hypertensive patients need to carefully control blood pressure, hypertension and kidney damage to a certain extent, can influence each other, together to increase the development of the disease. Renal blood vessels once blocked or hardened, renal blood flow hardening, renal blood flow is relatively reduced, then decline in renal function, which constitutes a pathogen of hypertension nephropathy. So, we will ask what causes the causes of hypertension nephropathy:
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1, acute glomerulonephritis onset, failed to control, clinical symptoms and urinary protein persist, extended more than 1 year, and evolved into chronic nephritis.
2, in the past there is a comprehensive history of acute nephritis, after weeks or months of convalescence, clinical symptoms and abnormal urine disappeared, normal renal function. After a long period of time, due to upper respiratory tract or other infections or overwork, suddenly appear proteinuria, edema or hypertension and nephritis symptoms.
3, the history of nephritis in the past, due to upper respiratory tract or other infections, significant edema and a lot of proteinuria and other symptoms of nephrotic syndrome.

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4, in the past no history of nephritis, short-term proteinuria, progressive hypertension and renal insufficiency.
5, in the past no history of nephritis, often due to infection or fatigue after the emergence of hematuria and proteinuria, after short-term break soon to reduce or disappear. So repeated attacks, without obvious clinical symptoms.



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